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Brief, acute exposure of the eyes to selenium dioxide Acute toxicity tests of selenium dioxide were conducted for 96 to 336 hr in intermittent-flow bioassay systems using six species of freshwater fish. Results: Reported symptoms and signs include abdominal symptoms, such as vomiting, diarrhea, pain, and nausea, as well as garlic-like odor on the breath. Methods: The published literature on the acute toxicity of KW - Toxic. Longer-term Brittle Nails Selenium toxicity can cause changes to your nail beds The maximum permissible limit suggested by the WHO in drinking water is 10 ppb, while USEPA sets the limit at 50 ppb as well. In acute toxicity, ingested doses in the range 1100 mg selenium/kg b.w., blood levels above 300 g Se/L and urinary levels above 170 g Se/L were associated with mortality (Hadrup and Ravn Selenium dioxide may be prepared by oxidation of selenium by burning in air, by reaction with hydrogen peroxide, and by the dehydration of selenous acid. Acute toxicity tests showed that selenium dioxide was a strong antagonist against mercuric chloride toxicity in goldfish. 2 . The decreasing order of species sensitivity was: fathead minnow, flagfish, brook trout, channel catfish, goldfish, and bluegil. Acute toxicity tests showed that selenium dioxide was a strong antagonist against mercuric chloride toxicity in goldfish. KW - Selenate. The level of excessive supplementation leading to selenosis is contentious, a study from China suggested at least 850 g daily 1. Concentrations of selenium in blood and urine samples in non-fatal cases are close to those observed in fatal cases. Ethanol (96%) was obtained from Xilong Chemicals (China). Curves relating median lethal concentration to exposure time for each species exposed for more than 168 hr A 56-year-old man ingested 1.7g of sodium selenite (11mg Se/kg bw) and suffered from Industrial airborne Se compounds likely include elemental Se and selenium dioxide dusts and hydrogen selenide gas. Excessive simultaneous intake of vitamin C might have reduced selenite to elemental selenium and thus alleviated the toxic reaction ( Jensen, 1984 ). A 48-year-old woman took 2000 mg of selenium dioxide. She presented with mildly altered consciousness and hematemesis and mucosal damage throughout the oral cavity, esophagus, and stomach. The decreasing order of species The dermal LD 50 for selenium dioxide in rabbits is 4 mg kg 1 . The decreasing order of species sensitivity was: fathead minnow, flagfish, brook trout, channel catfish, goldfish, and bluegill. Upon contact with human skin, industrial selenium compounds have been reported to cause rashes, redness, heat, swelling, and pain. Selenium dioxide. DO - 10.1016/j.jtemb.2019.126435 In the absence of oxygen when the soil is acidic, the amount of biologically available selenium should be low. Elemental selenium that cannot dissolve in water and other insoluble forms of selenium (such as selenium sulfide and heavy metal selenides) are less mobile and will usually remain in the soil, posing less of a risk for exposure. Selenium is a naturally occurring substance that is toxic at high concentrations but is also a nutritionally essential element. Acute toxicity tests of selenium dioxide were conducted for 96 to 336 hr in intermittent-flow bioassay systems using six species of freshwater fish. Acute toxicity tests of selenium dioxide were conducted for 96 to 336 hr in intermittent-flow bioassay systems using six species of freshwater fish. Source: S&P GlobalPlatts Metals Week. 3 Annual average New York dealer price for 99.5% selenium. The mechanism by which selenium exerts toxic effects is unknown, but existing theories are discussed in Section 3.5. Curves relating median lethal concentration to exposure time for each species exposed for more than 168 hr U2 - 10.1016/j.jtemb.2019.126435. The poisonous nature of many selenium compounds remained more or less a laboratory curiosity until the 1930s, when it was discovered that selenium was the active principle in forages and grains that caused alkali disease in livestock raised in certain areas of the American great plains. KW - Gun bluing. The decreasing order of species sensitivity was: fathead minnow, flagfish, brook trout, channel catfish, goldfish, and bluegil. Background: Although selenium is an essential element for humans, acute toxicity has been reported after high oral exposure. Acute toxicity tests of selenium dioxide were conducted for 96 to 336 hr in intermittent-flow bioassay systems using six species of freshwater fish. KW - Acute poisoning. Selenosis may result if the daily selenium intake exceeds 400-700 g per day (cf. Reduction of selenium dioxide to elemental selenium is responsible for the selenium toxicity treating effect of Paradoxically, whole body accumulations of total mercury were found to be significantly increased with the administration of Se. Concentrations of selenium in blood and urine samples in non-fatal cases are close to those observed in fatal cases. Diagnosis of selenium toxicity is usually clinical; sometimes blood glutathione peroxidase is measured. Nanoscale selenium has lower toxicity, higher biological activity [8, 17], and lower cytotoxicity than bulk selenium . The decreasing order of species At high doses ( > 900 mcg/day), selenium causes toxicity. Inorganic oxidizing agents, such as SELENIUM DIOXIDE, react with reducing agents to generate heat and products that may be flammable, combustible, or otherwise reactive. Average mass 110.959 Da. Abstract BACKGROUND: Although selenium is an essential element for humans, acute toxicity has been reported after high oral exposure. Monoisotopic mass 111.906349 Da. Selenium dioxide is toxic if inhaled and swallowed; danger of cumulative effects; very toxic to aquatic organisms causing long-term adverse effects to aquatic environments. 2.5. Paradoxically, whole body accumulations of total mercury were Keywords: Se; selenate; selenite; selenium dioxide; gun bluing; toxic; acute poisoning . Includes selenium metal and the selenium content of selenium dioxide. Mortality is a risk of acute selenium poisoning. 2. Selenite. Acute toxicity tests of selenium dioxide were conducted for 96 to 336 hr in intermittent-flow bioassay systems using six species of freshwater fish. Long-term exposure to selenium, sodium selenite, sodium selenate, or selenium dioxide may cause paleness, coated tongue, stomach disorders, nervousness, metallic taste The selenium toxicity in drinking water has caused debate since the 1970s. Food. Hydrogen selenide is the most acutely toxic selenium compound. Acute (short-term) exposure to elemental selenium, hydrogen selenide, and selenium dioxide by inhalation results primarily in respiratory effects, such as irritation of the mucous membranes, pulmonary edema, severe bronchitis, and bronchial pneumonia. KW - Selenite. KW - Selenium dioxide. SELENIUM This may be a conservative value for selenium compounds in general since it is based on sodium selenite, which is orders of magnitude more toxic than many other selenium compounds. Molecular Formula OSe. KW - Se. Selenium dioxide (99.99%) and 3,3-diaminobenzidine (99.8%) were purchased from Sigma-Aldrich. Mortality is a risk of acute selenium poisoning. Brief exposures to high levels of elemental selenium or selenium dioxide in air can result in respiratory tract irritation, bronchitis, difficulty breathing, and stomach pains. Selenium dioxide is highly to moderately toxic, with oral LD 50 values in rodents from 20 to 70 mg kg 1. METHODS: The published literature on the acute toxicity of oral selenium was gathered and Background: Although selenium is an essential element for humans, acute toxicity has been reported after high oral exposure. Toxicity Data: Selenium dioxide LD50 (oral, rat) 68 mg/kg LD50 (subcutaneous, rabbit) 4mg/kg Selenium dioxide is listed by the IARC as Group 3, Unclassifiable (as Selenium and selenium Selenium dioxide is the active ingredient in some cold-bluing solutions. It was also used as a toner in photographic developing . Selenium is an essential element, but ingestion of more than 5 mg/day leads to nonspecific symptoms. Elemental selenium can be oxidized to form selenium dioxide. Hydrogen selenide is the most acutely toxic selenium Human toxicity from environmental exposure to elemental selenium and selenium salts is rare. Exposure to selenious acid or to selenium dioxide can cause serious toxicity (Barceloux, Non-fatal human acute toxicity following the ingestion of selenium 3.1. Oral LD 50 values for The practical nature of this problem stimulated a great deal of research on both chronic and The decreasing order of species sensitivity was: fathead minnow, flagfish, brook trout, channel catfish, goldfish, and bluegill. In adults, Selenium dioxide monohydrate deficiency states resulting from long-term TPN support, Selenium dioxide monohydrate as selenomethionine or selenious acid, administered intravenously at 100 mcg/day for a period of 24 and 31 days, respectively, has been reported to reverse deficiency symptoms without toxicity. Revised IDLH: 1 mg Se/m 3 Basis for revised IDLH: The revised IDLH for selenium compounds is 1 mg Se/m 3 based on acute toxicity data in animals [Olson 1986; Pletnikova 1970a, 1970b]. The acute toxicity associated with oral selenium ingestion and the blood and urinary levels of selenium in different cases of poisonings were reviewed. advice from the National Academy of Science in the US that adult males should aim for 4070 g and females 4555 g per day). Acute toxicity tests of selenium dioxide were conducted for 96 to 336 hr in intermittent-flow bioassay systems using six species of freshwater fish. Selenium toxicity comprising of ocular pain, nail bed burns and painful skin can be treated with an ointment or solution of sodium thiosulfate (10%). ChemSpider ID 22440. In humid conditions selenium dioxide takes transformation into selenious acid. 392 Adverse effects after occupational exposures to Se dusts or Se Methods: The published literature on the acute toxicity of oral selenium was gathered and reviewed.

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